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application opening date: 2024-04-02T09:00:00+01:00
organization part: Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM)
type: Vacancy
comment: We are seeking an enthusiastic Research Assistant to work within a vibrant research team headed by Professor David Hodson at the Oxford Centre for Diabetes Endocrinology and Metabolism (OCDEM) on studies related to type 1 diabetes (https://www.rdm.ox.ac.uk/people/david-hodson). The postholder will contribute to a Steve Morgan Foundation Type 1 Diabetes Grand Challenge, which is led by Oxford together with partners at the University of Birmingham and FMP Leibniz Berlin. The overarching aims of the Grand Challenge are to: 1) produce better functioning stem cell-derived beta cells to ensure long-lasting insulin-independence in people living with type 1 diabetes (T1D); 2) precision deliver therapy to the pancreatic beta cell surface that can fend off immune attack in the early stages of T1D; and 3) coax alpha cells, which survive immune destruction during T1D, to convert into insulin-positive cells to provide a new supply of beta cells without the need for transplantation. To allow this, we will target two surface receptors, which are highly expressed by alpha cells and beta cells. The major goal is to help improve stem cell-derived beta cell survival and function, allow immunotherapy to work better for longer with fewer side effects, and provide more evidence that endogenous beta cells can be regenerated, together making beta cell replacement a possibility for more people living with T1D. Although full training will be given in a variety of techniques, the person appointed will be expected to work independently on most tasks or in collaboration with the support of colleagues. Methods will include cell culture, stem cell-derived beta cell generation, hormone assays, sequencing, cloning and optical imaging. The position would suit a cell biologist with experience of imaging, molecular genetics and cell culture. A background in diabetes, metabolism or neuroscience is desirable, but not essential, as full support and training will be provided. The position, funded by a Ste…
notation: 172064
salary: Standard Grade 6: £32,332 - £38,205 per annum
further particulars: 172064 - JD Research Assistant
application closing date: 2024-04-30T12:00:00+01:00
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value: Oxford Centre for Diabetes, Endocrinology and Metabolism Summary Job title Research Assistant Division Medical Sciences Division Department Radcliffe Department of Medicine (RDM) - Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM) Location OCDEM, Churchill Hospital, Headington, Oxford, OX3 7LE Grade and salary Grade 6: £32,332 - £38,205 per annum Hours Full time Contract type Fixed-term until 28/02/2029 Reporting to Professor David Hodson Vacancy reference 172064 Additional information Interviews are scheduled to be held on 22/05/2024 Research topic Type 1 diabetes Principal Investigator / supervisor Professor David Hodson Project team Project web site https://www.rdm.ox.ac.uk/people/david-hodson Funding partner The funds supporting this research project are provided by the “Steve Morgan Foundation Type 1 Diabetes Grand Challenge” by Diabetes UK and Steve Morgan Foundation. Recent publications doi: 10.1021/jacsau.2c00130 10.1038/s41467-022-35716-1 doi: 10.1172/jci.insight.140288 doi: 10.1038/s41467-020-20632-z 10.1016/j.celrep.2020.107761 doi: 10.1038/s41467-020-14309-w The role The successful applicant will join Prof. David Hodson’s research group at the Oxford Centre for Diabetes, Endocrinology and Metabolism, University of Oxford. The lab has recently been awarded Steve Morgan Foundation Type 1 Diabetes Grand Challenge funding, together with partners at the University of Birmingham (Dr Ildem Akerman) and FMP Leibniz Berlin (Dr Johannes Broichhagen). The overarching aims of our Grand Challenge are to: 1) produce better functioning stem cell-derived beta cells to ensure longlasting insulin-independence following transplantation into people living with T1D; 2) precision deliver therapy to the pancreatic beta cell surface that can fend off immune attack in the early stages of T1D; and 3) coax alpha cells, which survive immune destruction during T1D, to convert into insulin-positive cells to provide a new supply of beta cells without the need for transp…

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